Hostname: page-component-586b7cd67f-2plfb Total loading time: 0 Render date: 2024-11-24T12:42:00.092Z Has data issue: false hasContentIssue false
  • English
  • Français

Acriflavin resistant rII deletions of bacteriophage T4

Published online by Cambridge University Press:  14 April 2009

David H. Parma
David H. Parma
Affiliation:
Department of Genetics, University of Washington, Seattle, Washington
Rights & Permissions [Opens in a new window]

Extract

Core share and HTML view are not available for this content. However, as you have access to this content, a full PDF is available via the ‘Save PDF’ action button.

The extent and phenotype of acriflavin-resistant rII deletions have been examined. The properties of these deletions confirm that acriflavin resistance may result from a loss of function at the ac locus and that the ac locus coincides with the rII distal portion of the dispensable region which is adjacent to the rIIB cistron.

Type
Short Papers
Information
Genetics Research , Volume 13 , Issue 3 , June 1969 , pp. 329 - 331
Copyright
Copyright © Cambridge University Press 1969

References

REFERENCES

Bautz, F. A. & Bautz, E. K. F. (1967). Mapping of deletions in a non-essential region of the phage T4 genome. J. molec. Biol. 28, 345–356.CrossRefGoogle Scholar
Dove, W. F. (1968). The extent of rII deletions in phage T4. Genet. Res. Camb. 11, 215–219.CrossRefGoogle ScholarPubMed
Edgar, R. S. & Epstein, R. H. (1961). Inactivation by ultraviolet light of an acriflavinsensitive gene function in phage T4D. Science, N.Y. 134, 327–328.CrossRefGoogle ScholarPubMed
Goldberg, E. (1966). The amount of DNA between genetic markers in phage T4. Proc. natn. Acad. Sei. U.S.A. 56, 1457–1463.CrossRefGoogle ScholarPubMed
Parma, D. H. (1968). Genetic studies on chromosome structure and recombination in bacteriophage T4D. Doctoral dissertation, Department of Genetics, University of Washington, Seattle, Washington.Google Scholar