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Hyperglycemia, Lumbar Plexopathy and Hypokalemic Rhabdomyolysis Complicating Conn's Syndrome

Published online by Cambridge University Press:  18 September 2015

Chi-Ping Chow ,
Christopher J. Symonds  and
Douglas W. Zochodne
Chi-Ping Chow
Affiliation:
Department of Clinical Neurosciences and The Neuroscience Research Group,The University of Calgary, Calgary.
Christopher J. Symonds
Affiliation:
Department of Clinical Neurosciences and The Neuroscience Research Group,The University of Calgary, Calgary.
Douglas W. Zochodne*
Affiliation:
Department of Clinical Neurosciences and The Neuroscience Research Group,The University of Calgary, Calgary.
*
The University of Calgary, Department of Clinical Neurosciences, Room 182A, 3330 Hospital Drive N.E., Calgary, Alberta, Canada T2N 4N1
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Abstract:

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Background:

Lumbosacral plexopathy is a complication of diabetes mellitus. Conn's syndrome from an aldosterone secreting adenoma may be associated with hypokalemia and rhabdomyolysis but mild hyperglycemia also usually occurs.

Methods:

Case description.

Results:

A 70-year-old male diagnosed as having Conn's syndrome, hypokalemia and mild hyperglycemia developed rhabdomyolysis and lumbar plexopathy as a presenting feature of his hyperaldosteronism. His rhadbdomyolysis rapidly cleared following correction of hypokalemia but recovery from the plexopathy occurred slowly over several months. Definite resection of the aldosterone secreting adenomas reversed the hyperglycemia.

Conclusion:

Our patient developed lumbar plexopathy resembling that associated with diabetes mellitus despite the presence of only mild and transient hyperglycemia.

Résumé:

RÉSUMÉ:Introduction:

La plexopathie lombosacrée est une complication du diabète. Le syndrome de Conn dû â un adénome sécréfant de l'aldostérone peut étre associe a une hypokaliemie et a un rhabdomyolyse, mais une legere hyperglycemic peut ussi etre présente.

Méthodes:

Description d'un cas.

Résultats:

Un patient âgè de 70 ans porteur d'un syndrome de Conn, d'une hypokaliémie et d'une légère hyperglycémic a développé une rhabdomyolyse et une plexopathie lombaire comme manifestation initiale de son hyperaldostéronisme. La rhabdomyolyse s'est résolue rapidement suite à la correction de l'hypokaliémie, mais la guérison de sa plexopathie a pris plusieurs mois. La résection définitive de l'adénome sécréfant de l'aldostérone a fait disparaître l'hyperglycémie.

Conclusions:

Notre patient a développé une plexopathie lombosacrée ressemblant à une plexopathie diabétique bien que son hyperglycémic n'ait été que légère et transitoire.

Type
Original Articles
Information
Canadian Journal of Neurological Sciences , Volume 24 , Issue 1 , February 1997 , pp. 67 - 69
Copyright
Copyright © Canadian Neurological Sciences Federation 1997

References

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