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Delinquent peer affiliation as an etiological moderator of childhood delinquency

Published online by Cambridge University Press:  20 January 2012

S. A. Burt*
Affiliation:
Department of Psychology, Michigan State University, East Lansing, MI, USA
K. L. Klump
Affiliation:
Department of Psychology, Michigan State University, East Lansing, MI, USA
*
*Address for correspondence: S. A. Burt, Ph.D., Department of Psychology, Michigan State University, 107D Psychology Building, East Lansing, MI 48824, USA. (Email: [email protected])

Abstract

Background

Prior research has indicated that affiliation with delinquent peers activates genetic influences on delinquency during adolescence. However, because other studies have indicated that the socializing effects of delinquent peers vary dramatically across childhood and adolescence, it is unclear whether delinquent peer affiliation (DPA) also moderates genetic influences on delinquency during childhood.

Method

The current study sought to evaluate whether and how DPA moderated the etiology of delinquency in a sample of 726 child twins from the Michigan State University Twin Registry (MSUTR).

Results

The results robustly supported etiological moderation of childhood delinquency by DPA. However, this effect was observed for shared environmental, rather than genetic, influences. Shared environmental influences on delinquency were found to be several-fold larger in those with higher levels of DPA as compared to those with lower levels. This pattern of results persisted even when controlling for the overlap between delinquency and DPA.

Conclusions

Our findings bolster prior work in suggesting that, during childhood, the association between DPA and delinquency is largely (although not solely) attributable to the effects of socialization as compared to selection. They also suggest that the process of etiological moderation is not specific to genetic influences. Latent environmental influences are also amenable to moderation by measured environmental factors.

Type
Original Articles
Copyright
Copyright © Cambridge University Press 2012 

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