Hostname: page-component-78c5997874-xbtfd Total loading time: 0 Render date: 2024-11-03T08:28:02.158Z Has data issue: false hasContentIssue false

Characterisation of 3-beta-hydroxysteroid dehydrogenase from pig liver and testis in relation to boar taint

Published online by Cambridge University Press:  23 November 2017

S. I. Nicolau-Solano*
Affiliation:
Department of Clinical Veterinary Science, School of Veterinary Sciences, University of Bristol, Langford, Bristol, BS40 5DU, U.K.
F. M. Whittington
Affiliation:
Department of Clinical Veterinary Science, School of Veterinary Sciences, University of Bristol, Langford, Bristol, BS40 5DU, U.K.
J. D. Wood
Affiliation:
Department of Clinical Veterinary Science, School of Veterinary Sciences, University of Bristol, Langford, Bristol, BS40 5DU, U.K.
E. Doran
Affiliation:
Department of Clinical Veterinary Science, School of Veterinary Sciences, University of Bristol, Langford, Bristol, BS40 5DU, U.K.
Get access

Extract

Boar taint is a major meat quality defect, which affects about 10% of entire male pigs. It is due to an excessive accumulation of skatole and androstenone in adipose tissue. One of the reasons for accumulation of these compounds is a low rate of their metabolism. Androstenone is metabolised in liver via the enzyme 3-beta-hydroxysteroid dehydrogenase (HSD). This enzyme is well characterised in the testis, where it participates in the synthesis of steroids, while its properties in liver are unknown. The aim of the present study was to characterise and compare properties of HSD from pig liver versus pig testis when metabolising androstenone.

Type
Poster Presentations
Copyright
Copyright © The British Society of Animal Science 2005

Access options

Get access to the full version of this content by using one of the access options below. (Log in options will check for institutional or personal access. Content may require purchase if you do not have access.)