Crossref Citations
This article has been cited by the following publications. This list is generated based on data provided by
Crossref.
Fischetti, V. A.
Tsai, Pei-Jane
Lin, Yee-Shin
Kuo, Chih-Feng
Lei, Huan-Yao
and
Wu, Jiunn-Jong
1999.
Group A Streptococcus Induces Apoptosis in Human Epithelial Cells.
Infection and Immunity,
Vol. 67,
Issue. 9,
p.
4334.
Ginsburg, Isaac
Ward, Peter A
and
Varani, James
1999.
Can we learn from the pathogenetic strategies of group A hemolytic streptococci how tissues are injured and organs fail in post-infectious and inflammatory sequelae?.
FEMS Immunology & Medical Microbiology,
Vol. 25,
Issue. 4,
p.
325.
Sansonetti, Philippe
2000.
Bactéries et apoptose.
Annales de l'Institut Pasteur / Actualités,
Vol. 11,
Issue. 4,
p.
65.
Diez, Eduardo
Yaraghi, Zahra
MacKenzie, Alex
and
Gros, Philippe
2000.
The Neuronal Apoptosis Inhibitory Protein (Naip) Is Expressed in Macrophages and Is Modulated After Phagocytosis and During Intracellular Infection withLegionella pneumophila.
The Journal of Immunology,
Vol. 164,
Issue. 3,
p.
1470.
Russell, David G.
2000.
Virulence Mechanisms of Bacterial Pathogens.
p.
105.
Martínez, José L.
and
Baquero, Fernando
2002.
Interactions among Strategies Associated with Bacterial Infection: Pathogenicity, Epidemicity, and Antibiotic Resistance.
Clinical Microbiology Reviews,
Vol. 15,
Issue. 4,
p.
647.
Splettstoesser, Wolf D.
and
Schuff‐Werner, Peter
2002.
Oxidative stress in phagocytes—“The enemy within”.
Microscopy Research and Technique,
Vol. 57,
Issue. 6,
p.
441.
Evans, Stephen M.
Ashwood, Paul
Warley, Alice
Berisha, Fatmire
Thompson, Richard P.H.
and
Powell, Jonathan J.
2002.
The role of dietary microparticles and calcium in apoptosis and interleukin-1β release of intestinal macrophages.
Gastroenterology,
Vol. 123,
Issue. 5,
p.
1543.
Hazlett, Linda Dondero
2004.
Corneal response to Pseudomonas aeruginosa infection.
Progress in Retinal and Eye Research,
Vol. 23,
Issue. 1,
p.
1.
Molmeret, Maëlle
Bitar, Dina M.
Han, Lihui
and
Kwaik, Yousef Abu
2004.
Disruption of the Phagosomal Membrane and Egress ofLegionella pneumophilainto the Cytoplasm during the Last Stages of Intracellular Infection of Macrophages andAcanthamoeba polyphaga.
Infection and Immunity,
Vol. 72,
Issue. 7,
p.
4040.
Schmid-Hempel, Paul
2005.
Natural insect host-parasite systems show immune priming and specificity: puzzles to be solved.
BioEssays,
Vol. 27,
Issue. 10,
p.
1026.
Fortier, Anne
Diez, Eduardo
and
Gros, Philippe
2005.
Naip5/Birc1e and susceptibility to Legionella pneumophila.
Trends in Microbiology,
Vol. 13,
Issue. 7,
p.
328.
EVI SIMOVART, HELLE
AREND, ANDRES
TAPFER, HELLE
KOKK, KERSTI
AUNAPUU, MARINA
POLDOJA, ELLE
SELSTAM, GUNNAR
and
LIIGANT, AADE
2006.
Experimental Sepsis.
Annals of the New York Academy of Sciences,
Vol. 1090,
Issue. 1,
p.
253.
Jiang, Weiwen
Bell, Charles W.
and
Pisetsky, David S.
2007.
The Relationship between Apoptosis and High-Mobility Group Protein 1 Release from Murine Macrophages Stimulated with Lipopolysaccharide or Polyinosinic-Polycytidylic Acid.
The Journal of Immunology,
Vol. 178,
Issue. 10,
p.
6495.
2008.
Medical Toxicology of Natural Substances.
p.
150.
Bellingan, Geoffrey J.
and
Laurent, Geoffrey J.
2008.
The Resolution of Inflammation.
p.
75.
Rajalingam, Krishnaraj
Sharma, Manu
Lohmann, Christine
Oswald, Monique
Thieck, Oliver
Froelich, Christopher J.
Rudel, Thomas
and
Valdivia, Raphael H.
2008.
Mcl-1 Is a Key Regulator of Apoptosis Resistance in Chlamydia trachomatis-Infected Cells.
PLoS ONE,
Vol. 3,
Issue. 9,
p.
e3102.
Wang, Jia-he
Zhou, Yi-jun
Tian, Li
and
He, Ping
2009.
Phosphoinositide 3-Kinase/Akt and Nuclear Factor-κB Are Involved in Staphylococcus Aureus-induced Apoptosis in U937 Cells.
Chinese Medical Sciences Journal,
Vol. 24,
Issue. 4,
p.
231.
Sharma, Manu
and
Rudel, Thomas
2009.
Apoptosis resistance inChlamydia-infected cells: a fate worse than death?.
FEMS Immunology & Medical Microbiology,
Vol. 55,
Issue. 2,
p.
154.
Gao, Xueli
Raghavamenon, Achuthan C.
D’Auvergne, Oswald
and
Uppu, Rao M.
2009.
Cholesterol secoaldehyde induces apoptosis in J774 macrophages via mitochondrial pathway but not involving reactive oxygen species as mediators.
Biochemical and Biophysical Research Communications,
Vol. 389,
Issue. 2,
p.
382.