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The relation between rates of growth and rickets in sheep on diets deficient in vitamin D.

Published online by Cambridge University Press:  27 March 2009

John Duckworth ,
William Godden  and
William Thomson
John Duckworth
Affiliation:
The Rowett Research Institute, Aberdeen
William Godden
Affiliation:
The Rowett Research Institute, Aberdeen
William Thomson
Affiliation:
The Rowett Research Institute, Aberdeen
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Extract

The blood picture for Ca, inorganic P and phosphatase during the development of rickets caused by uncomplicated vitamin D deficiency in sheep has been established. The earliest and most distinctive change was found to be in the serum Ca level.

Lambs were shown to have sufficient reserves of vitamin D to protect them against avitaminosis D for a period of about 6 weeks as judged by serum Ca levels. Rickets did not develop unless there was a moderate degree of growth and, in such cases, the serum Ca level was below 7 mg./100 ml.

The practical application of the results was discussed.

The authors desire to acknowledge the co-operation of Dr A. H. H. Fraser in the earlier planning of this work.

Type
Research Article
Information
The Journal of Agricultural Science , Volume 33 , Issue 4 , October 1943 , pp. 190 - 196
Copyright
Copyright © Cambridge University Press 1943

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References

REFERENCES

Auchinachie, D. W. & Emslie, A. R. G. (1933). Biochem. J. 27, 351.CrossRefGoogle Scholar
Auchinachie, D. W. & Fraser, A. H. H. (1932). J. Agric. Sci. 22, 560.CrossRefGoogle Scholar
Bodansky, A. (1932). J. Biol. Chem. 99, 197.CrossRefGoogle Scholar
Bodansky, A. (1933). J. Biol. Chem. 101, 93.CrossRefGoogle Scholar
Boelter, M. D. D. & Greenberg, D. M. (1941). J. Nutrit. 21, 61.CrossRefGoogle Scholar
Carnes, W. H.Pappenheimer, A. M. & Stoerk, H. C. (1942). Proc. Soc. Exp. Biol., N.Y., 51, 314.CrossRefGoogle Scholar
Clark, E. P. & Collip, J. B. (1925). J. Biol. Chem. 63, 461.CrossRefGoogle Scholar
Elliot, W. & Crichton, A. (1926). J. Agric. Sci. 16, 65.CrossRefGoogle Scholar
Fiske, C. H. & Subbarow, Y. (1925). J. Biol. Chem. 66, 375.CrossRefGoogle Scholar
Franklin, M. C. (1934–1935). 4th Rep. of the Director, Inst. Anim. Pathol. Univ. Camb. p. 111.Google Scholar
Godden, W. & Duckworth, J. (1935). Biochem. J. 29, 445.CrossRefGoogle Scholar
Guilbert, H. R., Miller, R. F. & Hughes, E. H. (1937). J. Nutrit. 13, 543.CrossRefGoogle Scholar
Ham, A. W., Littner, N., Drake, T. G. H., Robertson, E. C. & Tisdall, F. F. (1940). Amer. J. Pathol. 16, 277.Google Scholar
Hamilton, B. & Schwartz, C. (1932). J. Pharmacol. 46, 285.Google Scholar
Hart, E. B. (1940–1941). Nutrit. Abstr. Rev. 10, 261.Google Scholar
Kay, H. D. (1930). J. Biol. Chem. 89, 235.CrossRefGoogle Scholar
Marston, H. R. (1935). J. Coun. Sci. Industr. Res. Austr. 8, 293.Google Scholar
Mitchell, H. H. & McClure, F. J. (1937). Bull. Nat. Res. Coun., Wash., no. 99.Google Scholar
Stewart, J. (1932–1933). 2nd Rep. of the Director, Inst. Anim. Pathol. Univ. Camb. p. 58.Google Scholar
Stewart, J. (1934–1935). 4th Rep. of the Director, Inst. Anim. Pathol. Univ. Camb. p. 179.Google Scholar