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The transcobalamin 776C>G polymorphism may be a modifiable genetic risk factor for Alzheimer's disease

Published online by Cambridge University Press:  29 June 2005

HENRIK ZETTERBERG ,
EBBA NEXÖ ,
LENNART MINTHON ,
ROBERTA BOSON ,
BJÖRN REGLAND ,
ANDREW MCCADDON  and
K.A.J. BLENNOW
HENRIK ZETTERBERG
Affiliation:
Institutes of Clinical Neuroscience, Sahlgrenska University Hospital, Göteborg University, Sweden. Email: [email protected] Institutes of Laboratory Medicine, Sahlgrenska University Hospital, Göteborg University, Sweden. Email: [email protected] Center for Neurologic Diseases, Harvard Medical School, Boston, U.S.A.
EBBA NEXÖ
Affiliation:
Department of Clinical Biochemistry, Aarhus University Hospital, Aarhus, Denmark
LENNART MINTHON
Affiliation:
Neuropsychiatric Clinic, Malmö University Hospital, S-205 02 Malmö, Sweden
ROBERTA BOSON
Affiliation:
Neuropsychiatric Clinic, Malmö University Hospital, S-205 02 Malmö, Sweden
BJÖRN REGLAND
Affiliation:
Institutes of Clinical Neuroscience, Sahlgrenska University Hospital, Göteborg University, Sweden. Email: [email protected]
ANDREW MCCADDON
Affiliation:
University of Wales College of Medicine, General Practice, Wrexham, U.K.
K.A.J. BLENNOW
Affiliation:
Institutes of Clinical Neuroscience, Sahlgrenska University Hospital, Göteborg University, Sweden. Email: [email protected] Institutes of Laboratory Medicine, Sahlgrenska University Hospital, Göteborg University, Sweden. Email: [email protected]
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Abstract

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Type
Letter to the Editor
Information
International Psychogeriatrics , Volume 17 , Issue 2 , June 2005 , pp. 329 - 331
Copyright
© International Psychogeriatric Association 2005