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The suprarenal glands in diphtheria

Published online by Cambridge University Press:  15 May 2009

Alex. Maclean
Affiliation:
Clinical Medical Officer, Glasgow Public Health Department; formerly Assistant Physician, Ruchill Fever Hospital, Glasgow
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In recent years, it has become apparent that lesions of the suprarenal glands occur more often in human disease than had formerly been suspected. The evident effect of such lesions is the production of profound general disturbances, which are seen in a chronic phase if the morbid process in the suprarenal tissue is slowly progressive, as, for example, in the ordinary case of Addison's disease, and in an acute phase if the lesion is of sudden onset, as in suprarenal haemorrhage, or if chronically diseased suprarenal tissue is subjected to sudden excessive demands, as in the critical stage of Addison's disease. In both phases, there is evidence to show that the best measures to employ in combating these disturbances, whether for prophylaxis or for therapy, consist of the administration to the patient of an extract of suprarenal cortex or, alternatively, of sodium chloride.

Type
Research Article
Copyright
Copyright © Cambridge University Press 1937

References

REFERENCES

Addison, (1903). A collection of the published writings of the late Thomas Addison, Practitioner, 71, 472.Google Scholar
Bbgg, & Harries, (1935). Lancet, i, 480.CrossRefGoogle Scholar
Benn, , Hughes, & Alstead, (1932). Lancet, i, 281.CrossRefGoogle Scholar
Brems, (1932). Ada Med. Scand., 14 Dec., p. 1.CrossRefGoogle Scholar
Britton, & Silvette, (1933). Amer. J. Physiol. 104, 399.Google Scholar
Britton, & Silvette, (1934). Amer. J. Physiol. 107, 190.CrossRefGoogle Scholar
Brown-SÉquard, . Quoted by Rabinowitz, (1923). Amer. J. Med. Sci. 166, 516.Google Scholar
Burton, & Chalmers, (1930). Lancet, i, 296.CrossRefGoogle Scholar
Donato, (1930). Thèse de Paris, No. 323.Google Scholar
Habbop, , Weinstein, , Soffer, & Trescher, (1933). J. Exp. Med. 58, 1.Google Scholar
Hartman, , Brownell, & Lockwood, (1932). Endocrinology, 16, 521.CrossRefGoogle Scholar
Herbert, & Bourne, (1931). Brit. Med. J. i, 94.CrossRefGoogle Scholar
Keb, C. B.Infectious Diseases (revised by Rundle, 1929).Google Scholar
Lereboullet, Gourmay & Donato, (1931). Ann. de Méd., 05, p. 547.Google Scholar
Lucke, Wight & Kime, (1919). Arch. Int. Med. 24, 19.CrossRefGoogle Scholar
Maclagan, & Cooke, (1916). Lancet, ii, 1054.CrossRefGoogle Scholar
Maclean, (1936). Lancet, ii, 129.CrossRefGoogle Scholar
Muir, R. (1924). Text-Book of Pathology.Google Scholar
Pearl, & Brunn, (1928). Surg., Gyn. & Obstet., 09, p. 393.Google Scholar
Peters, & Gunn, (1930). J. Hygiene, 30, 420.Google Scholar
Peters, & Van Slyke, (1932). Quantitative Clinical Chemistry, vol. ii, pp. 737, 751, 835.Google Scholar
Polandowski, (1932). Thèse de Paris, No. 35.Google Scholar
Rabinowitz, (1923). Amer. J. Med. Sci. 166, 516.CrossRefGoogle Scholar
Rolleston, J. D. (1925). Acute Infectious Diseases.Google Scholar
Rowntree, (1933). Article on Addison's disease in Text-Book of Medicine by American Authors, 3rd ed., edited by Cecil, Russell L., p. 1249.Google Scholar
Rubinztejn, (1934). Thèse de Paris, No. 134.Google Scholar
Schwentker, & Noel, (1930). Bull. Johns Hopkins Hosp., 06, p. 358.Google Scholar
Thaddea, (1935). Klin. Woch., 7 09 (Annotation in Brit. Med. J. 1935, ii, 1003.)Google Scholar
Thomson, John (1925). The Clinical Study and Treatment of Sick Children, 4th ed., p. 461.Google Scholar