Hostname: page-component-78c5997874-dh8gc Total loading time: 0 Render date: 2024-11-08T10:31:46.503Z Has data issue: false hasContentIssue false
  • English
  • Français

Extracorporeal membrane oxygenation as a bridge to surgical treatment of flail tricuspid valve in a neonate

Published online by Cambridge University Press:  18 November 2005

Cammon B. Arrington ,
Peter C. Kouretas  and
Christopher R. Mart
Cammon B. Arrington
Affiliation:
Department of Pediatric Cardiology/Cardiac Surgery, University of Utah, Salt Lake City, Utah, United States of America
Peter C. Kouretas
Affiliation:
Department of Pediatric Cardiology/Cardiac Surgery, University of Utah, Salt Lake City, Utah, United States of America
Christopher R. Mart
Affiliation:
Department of Pediatric Cardiology/Cardiac Surgery, University of Utah, Salt Lake City, Utah, United States of America
Get access Rights & Permissions [Opens in a new window]

Abstract

A term infant rapidly developed profound cyanosis and metabolic acidosis shortly after an uncomplicated vaginal delivery. Echocardiography identified a flail antero-superior leaflet of the tricuspid valve, which was producing severe tricuspid insufficiency. The clinical state deteriorated despite maximal medical management, and the patient was placed on venoarterial extracorporeal membrane oxygenation. Within twenty-four hours, the metabolic acidosis corrected, inotropic support was discontinued, and the patient was weaned to minimal ventilator settings. Successful repair of the tricuspid valve was performed two days later.

Keywords

Atrioventricular valveanterior papillary muscleextracorporeal membrane oxygenationcongenital heart disease
Type
Brief Report
Information
Cardiology in the Young , Volume 15 , Issue 6 , December 2005 , pp. 660 - 662
Copyright
© 2005 Cambridge University Press

Access options

Get access to the full version of this content by using one of the access options below. (Log in options will check for institutional or personal access. Content may require purchase if you do not have access.)

References

Rowe RD, Hoffman T. Transient myocardial ischemia of the newborn infant: a form of severe cardiorespiratory distress in full-term infants. J Pediatr 1972; 81: 243–250.Google Scholar
Bucciarelli RL, Nelson RM, Egan EA, Eitzman DV, Gessner IH. Transient tricuspid insufficiency of the newborn: a form of myocardial dysfunction in stressed newborns. Pediatrics 1977; 59: 330–337.Google Scholar
Reller MD, Rice MJ, McDonald RW. Tricuspid regurgitation in newborn infants with respiratory distress: Echo-Doppler study. J Pediatr 1987; 110: 760–764.Google Scholar
Donnelly WH, Bucciarelli RL, Nelson RM. Ischemic papillary muscle necrosis in stressed newborn infants. J Pediatr 1980; 96: 295–300.Google Scholar
Setzer E, Ermocilla R, Tonkin I, John E, Sansa M, Cassady G. Papillary muscle necrosis in a neonatal autopsy population: Incidence and associated clinical manifestations. J Pediatr 1980; 96: 289–294.Google Scholar
Alkalay AL, Ferry DA, Pepkowitz SH, Chou PJ, Oakes GK, Pomerance JJ. Critical tricuspid insufficiency due to papillary muscle rupture. AJDC 1988; 142: 753–755.Google Scholar
Pinto CA, Herdy GV, Ferrari AH, et al. Severe tricuspid valve insufficiency due to anterior papillary muscle infarction of the right ventricle secondary to neonatal hypoxia. Arq Bras Cardiol 1992; 58: 475–478.Google Scholar
Pennington DG, Swartz MT. Circulatory support in infants and children. Ann Thorac Surg 1993; 55: 233–237.Google Scholar
Marsh TD, Shelton Jr LW. Neonatal tricuspid insufficiency with abnormal tricuspid valve treated with extracorporeal membrane oxygenation (ECMO): possible extension of ECMO use. Am J Perinatol 1993; 10: 36–38.Google Scholar
Butler TJ, Yoder BA, Seib P, Lally KP, Smith VC. ECMO for left ventricular assist in a newborn with critical aortic stenosis. Pediatr Cardiol 1994; 15: 38–40.Google Scholar