We use cookies to distinguish you from other users and to provide you with a better experience on our websites. Close this message to accept cookies or find out how to manage your cookie settings.
To save content items to your account,
please confirm that you agree to abide by our usage policies.
If this is the first time you use this feature, you will be asked to authorise Cambridge Core to connect with your account.
Find out more about saving content to .
To save content items to your Kindle, first ensure [email protected]
is added to your Approved Personal Document E-mail List under your Personal Document Settings
on the Manage Your Content and Devices page of your Amazon account. Then enter the ‘name’ part
of your Kindle email address below.
Find out more about saving to your Kindle.
Note you can select to save to either the @free.kindle.com or @kindle.com variations.
‘@free.kindle.com’ emails are free but can only be saved to your device when it is connected to wi-fi.
‘@kindle.com’ emails can be delivered even when you are not connected to wi-fi, but note that service fees apply.
Individuals with the pervasive developmental disorder Asperger's syndrome (AS) are generally of average or above average intelligence and attend mainstream schools. Despite their intelligence, some of the characteristics and challenges associated with AS can impact upon the quality of life they experience at school. Although both males and females are diagnosed with AS, females have been underrepresented and unrepresented in much of the AS research. Consequently, much of what is known about the school experiences of students with AS is based on studies consisting predominately of male perspectives. This article discusses what adult females with AS and parents of females with AS currently attending school wish teachers understood about AS in females. Many of the experiences described by participants indicate there is a lack of understanding about AS, both generally and specifically, in relation to females. Implications of these findings and recommendations for educators and schools are provided, and directions for future research are outlined.
Comportment can be understood through analysis of its individual components: insight, judgment, self-awareness, social adaptation, and empathy. The case of Phineas Gage has served as the guiding compass towards our understanding of the prefrontal cortex as a region critical for comportment. Modern neuroimaging using the skull of Gage has shown bihemispheric prefrontal lesions involving the orbitofrontal cortex, the medial frontal cortex, and the anterior cingulate gyrus. A variety of diseases that preferentially affect the prefrontal cortex and that result in increased aggression, loss of empathy, and disinhibition have provided neurologists with insight into the brain structures responsible for comportment. This chapter discusses the pathogenesis of developmental disorders such as autism and Asperger's Syndrome (AS), degenerative processes such as frontotemporal dementia (FTD), physical injury to the prefrontal cortex, and schizophrenia as well as relevant functional neuroimaging studies.
This systematic review explores the psychosocial wellbeing of neurotypical (NT) women in intimate relationship with persons with Asperger's Syndrome (AS), and intervention models for such relationships. Over 20 pertinent scholarly databases were searched in April 2011. The initial search yielded 1,736 unique items. Articles were eligible if they were a peer-reviewed journal article or a peer-reviewed thesis investigating adult couple relationships where only one partner had AS. Ten studies were agreed by both reviewers to have met these final inclusion criteria. Utilising the National Health and Medical Research Council (NHMRC) guidelines the body of evidence comprising the final 10 studies was assessed to be of an overall grade D or ‘weak’. None of the studies included for final analyses were intervention studies.
Prior studies suggest that autism spectrum disorders (ASD) are associated with a domain-specific memory impairment for faces. The underlying cause of this problem and its relation to impaired visual scanning of faces—particularly of the eyes—remains to be determined. We recorded eye movements while 22 high-functioning ASD and 21 typically developing (TD) adolescents encoded and later recognized faces and objects from a single, nonsocial object category (electric fans). Relative to TD subjects, ASD individuals had poorer memory for faces, but not fans. Correlational analyses showed significant relationships between recognition memory and fixations. Eye tracking during encoding revealed that TD subjects made more fixations to faces than fans, whereas ASD individuals did not differ in number of fixations made to each stimulus type. Moreover, although both the TD and ASD groups showed a strong preference for fixating the eyes more than the mouth, the ASD subjects were less likely than TD subjects to scan regions of the face outside of the primary facial features (i.e., eyes, nose, and mouth). We concluded that ASD individuals have a domain-specific memory impairment for faces relative to mechanical objects and that this impairment may be related to abnormal scanning during encoding. (JINS, 2011, 17, 1021–1029)
Individuals with schizophrenia and individuals with high-functioning autism (HFA) seem to share some social, behavioral and biological features. Although marked impairments in social cognition have been documented in both groups, little empirical work has compared the social cognitive functioning of these two clinical groups.
Method
Forty-four individuals with schizophrenia, 36 with HFA and 41 non-clinical controls completed a battery of social cognitive measures that have been linked previously to specific brain regions.
Results
The results indicate that the individuals with schizophrenia and HFA were both impaired on a variety of social cognitive tasks relative to the non-clinical controls, but did not differ from one another. When individuals with schizophrenia were divided into negative symptom and paranoid subgroups, exploratory analyses revealed that individuals with HFA may be more similar, in terms of the pattern of social cognition impairments, to the negative symptom group than to the paranoia group.
Conclusions
Our findings provide further support for similarities in social cognition deficits between HFA and schizophrenia, which have a variety of implications for future work on gene–brain–behavior relationships.
Childe was a considerable influence on my archaeology, when I was an undergraduate at the Institute of Archaeology (London). His personality was enigmatic, as were parts of his publications. In this contribution some critical comments are made of his work, together with discussions on his Marxism, the Neolithic Revolution, what is meant by progress, and his all too brief consideration of the range of archaeological science. It will be argued that his inability to make close friendships, and other aspects of his personality, would suggest that he had Asperger's syndrome.
This controlled trial of a teacher training intervention aimed to increase teacher competence in managing the problem behaviours associated with Asperger's syndrome, as manifested in a classroom setting. All teacher-participants currently managed a student with Asperger's syndrome in an inclusive classroom setting. Measures were taken on two occasions: pre-workshop and 6-week follow-up. Variables of interest were number of problem behaviours, success of teacher strategies used to manage problem behaviours and teacher self-efficacy in managing behaviours. Qualitative data assessing both the utility of the workshop and effectiveness of the individual management strategies was also gathered. At 6-week follow up, teachers reported increased confidence in their ability to manage the student with Asperger's syndrome, fewer problem behaviours displayed by the student and increased success in using strategies to manage the student in the classroom. The utility of both the workshop itself and individual management strategies were also endorsed by all teacher-participants. Suggestions for future research and limitation of the study are also discussed.
Several prior reports have found that some young children with autism spectrum disorder [ASD; including autism and Asperger's syndrome and pervasive developmental disorder – not otherwise specified (PDD-NOS)] have a significant increase in head size and brain weight. However, the findings from older children and adults with ASD are inconsistent. This may reflect the relatively small sample sizes that were studied, clinical heterogeneity, or age-related brain differences.
Method
Hence, we measured head size (intracranial volume), and the bulk volume of ventricular and peripheral cerebrospinal fluid (CSF), lobar brain, and cerebellum in 114 people with ASD and 60 controls aged between 18 and 58 years. The ASD sample included 80 people with Asperger's syndrome, 28 with autism and six with PDD-NOS.
Results
There was no significant between-group difference in head and/or lobar brain matter volume. However, compared with controls, each ASD subgroup had a significantly smaller cerebellar volume, and a significantly larger volume of peripheral CSF.
Conclusions
Within ASD adults, the bulk volume of cerebellum is reduced irrespective of diagnostic subcategory. Also the significant increase in peripheral CSF may reflect differences in cortical maturation and/or ageing.
Asperger's syndrome (AS) as a diagnostic category has gained enormous popularity and the label is being applied with increasing liberality. While greater awareness of the disorder may be of advantage to certain children and their families, we argue that the over-inclusive use of the diagnosis may also lead to professional conflicts and ethical dilemmas. Perhaps most concerning of these is that diagnostic ‘generosity’ may exclude children from treatments they would have received if an alternative formulation had been considered. We present clinical scenarios, with special consideration of contextual and intergenerational influences on the children's early lives, and offer alternative conceptualisations. We argue that the new ‘epidemic’ of Asperger's should not be allowed to compromise our professional standards. Assessment should always include a detailed family and developmental history. Finally, we would be interested in feedback from clinicians working in this field.
The current study follows a recent paper reporting that individuals with autism were just as susceptible to visual illusions as those without autism (Ropar & Mitchell, 1999). The possibility that individual differences may account for the failure to replicate Happé's (1996) findings is explored by presenting a battery of visuospatial tasks thought to measure weak central coherence (embedded figures, block design, Rey complex figure test). Participants with autism were distinguished by relatively good performance on visuospatial tasks, though there was no superiority effect in those with Asperger's syndrome. Performance on the visuospatial battery did not significantly predict susceptibility to illusions in various participant groups, including those with autism and Asperger's syndrome. This suggests that perception of illusions and performance on visuospatial tasks may rely on different mechanisms. The implications for the theory of weak central coherence are discussed.
Neuroimaging studies in patients with early acquired damage will primarily seek to identify enhanced activations in regions outside a structural lesion. By comparison, imaging studies in developmental disorders will typically look for an absence or abnormality of activation that is assumed to reflect a particular cognitive or affective impairment. This chapter deals with evidence indicating vulnerability effects in non-human animals and human patients with early structural lesions. It discusses the interaction of vulnerability and compensatory events in developmental disorders, with exemplary focus on autism, developmental language impairment (DLI), and dyslexia. The chapter presents developmental disorders both in terms of the obvious effects of maturational vulnerability and in terms of the typically more elusive effects of compensatory reorganization. It focuses on autism and Asperger's syndrome. In the discussion presented here on bottom-up and top-down approaches, there are interesting parallels in the debates about developmental disorders of spoken and of written language.
Thirteen children and adolescents with diagnoses of Asperger syndrome (AS) were matched
with 13 nonautistic control children on chronological age and verbal IQ. They were tested on
their ability to recognize simple facial emotions, as well as facial emotions paired with
matching, mismatching, or irrelevant verbal labels. There were no differences between the
groups at recognizing simple emotions but the Asperger group performed significantly worse
than the control group at recognizing emotions when faces were paired with mismatching
words (but not with matching or irrelevant words). The results suggest that there are
qualitative differences from nonclinical populations in how children with AS process facial
expressions. When presented with a more demanding affective processing task, individuals
with AS showed a bias towards visual-verbal over visual-affective information (i.e., words
over faces). Thus, children with AS may be utilizing compensatory strategies, such as verbal
mediation, to process facial expressions of emotion.
A recent finding that individuals with autism are not susceptible to illusions has been
explained by Happé (1996) as a sign of “weak central coherence” at lower levels of
processing. We investigated the phenomenon with a more sophisticated measure. In
Experiment 1, individuals with autism, Asperger's syndrome, moderate learning difficulties,
and typical development adjusted certain comparison lines and circles to make them appear
to be the same size in four visual illusions. With a minor exception, the participants with
autism and Asperger's syndrome evinced a systematic bias in their judgements in the illusion
condition. The extent of this was no different from control participants. In a second
experiment, a similar finding was obtained in a task where participants made verbal
judgements about the stimuli. The results suggest that lower-level coherence in visual
processing in autism is intact.
A case of abnormal bereavement is described in a man with borderline learning disability and Asperger's syndrome. Bereavement issues and concurrent assessment of psychiatric illness in people with pervasive developmental disorders are discussed. Particular reference is made to relevant psychopathology and possible underlying psychodynamic mechanisms.