Glufosinate controls glyphosate-resistant Palmer amaranth, but growers struggle to make timely applications. XtendFlexTM cotton, resistant to dicamba, glufosinate, and glyphosate, may provide growers an option to control larger weeds. Palmer amaranth control and cotton growth, yield, and fiber quality were evaluated in a rescue situation created by delaying the first POST herbicide application. Treatments consisted of two POST applications of dicamba plus glufosinate, separated by 14 d, with the first application timely (0-d delay) or delayed 7, 14, 21, or 28 d. All treatments included a layby application of diuron plus MSMA. Palmer amaranth, 14 d after first POST, was controlled 99, 96, 89, 75, and 73% with 0-, 7-, 14-, 21-, or 28-d delays, respectively. Control increased following the second application, and the weed was controlled at least 94% following layby. Cotton yield decreased linearly as first POST application was delayed, with yield reductions ranging from 8 to 42% with 7- to 28-d delays. Delays in first POST application delayed cotton maturity but did not affect fiber quality.

Field research was conducted in Tennessee at Jackson in 1991 and at Milan in 1992 to compare the effect of MSMA and pyrithiobac on cotton development, yield, and quality. In separate treatments to different plots, pyrithiobac at 0.14 kg ai ha–1 did not affect development, yield, and quality of cotton. MSMA at 2.24 kg ai ha–1 decreased cotton plant internode length 10 to 15% and height by 15% 5 wk after late treatment both years. MSMA applied to 50 cm cotton reduced subsequent plant height in 1992, but other plant characteristics were not affected. In 1991, MSMA increased squares and decreased blooms and bolls for monopodia and sympodia position one and two, which suggested a delay in plant development. In 1991, mechanical harvest lint yields were decreased by MSMA at first harvest while increasing second harvest lint yields. However, in 1991 only MSMA applied late decreased total harvest lint yield 20%. Plant mapping data determined that the yield decrease was a result of decreased yields at sympodia positions one and two. Cotton seed arsenic analysis indicated that MSMA-late increased arsenic levels by ≈ 1 ppmw in 1991 for sympodia at position one and two compared to the untreated check, while position two contained the highest level of 1.8 ppmw.

Field experiments were conducted in Alabama from 1992 through 1994 to evaluate the potential of the methyl ester of bensulfuron applied at sublethal rates as a plant growth regulator for reducing plant height and boll rot in cotton. Bensulfuron at 0.017 and 0.034 g ai/ha or mepiquat chloride at 10 g ai/ha was applied POST alone at the pinhead square or early-bloom stage of cotton growth or sequentially at 0.017 followed by (fb) 0.017 g/ha, 0.034 fb 0.034 g/ha of bensulfuron and 5 fb 5, 10 fb 10, 10 fb 20, or 20 fb 20 g/ha of mepiquat chloride. Mepiquat chloride had no effect on yield in 1992 and 1994 but decreased yield when applied sequentially in 1993. Bensulfuron was generally detrimental to first position fruit retention, and it delayed maturity. Treatments that reduced plant height did not reduce boll rot. Bensulfuron treatments that reduced plant height also reduced yield; therefore, the potential for its use as a growth regulator in cotton appears limited.

Field experiments conducted from 1987 through 1991 at the Delta Branch Experiment Station, Stoneville, MS, determined the effect of postemergence topical applications of fluometuron and MSMA on cotton yield and fruiting. Herbicide treatments applied to cotton in the cotyledon to 1-leaf growth stage were 1.12 kg ai ha-1 fluometuron plus 0.25% by vol nonionic surfactant, 2.2 kg ai ha-1 MSMA, and a combination of 1.12 kg ha-1 fluometuron and 2.24 kg ha-1 MSMA. The commercial formulation of MSMA contained nonionic surfactant. Visual injury of cotton 14 d after treatment ranged from 14 to 28% for fluometuron, 9 to 26% for MSMA, and 22 to 34% for the combination. Seed cotton yield at the first harvest was reduced by all herbicide treatments except in 1987 and for DES 119 cotton in 1990. In 1990, yield of DES 119 cotton treated with MSMA and the combination was less than the untreated control for the second harvest interval. Cotton yield from herbicide treatments at later harvests was equal to or greater than the untreated control. Only in 1988 did all treatments reduce total seed cotton yield below the untreated control. Fiber quality was not adversely affected by the herbicides. Compared to the control, plant mapping indicated that the number of sympodial branches per plant was reduced by MSMA and by the combination for DPL 50 cotton in 1990. Averaged over 4 experiment years, herbicide treatments did not reduce total bolls per plant; percentage of bolls in first, second, or outer positions; highest sympodium with two bolls; or number of sympodium with bolls in the first or second position. However, MSMA and the combination increased node number of the first sympodia by one and 1.5 positions, respectively, when compared to the control, indicating delayed maturity.

Field experiments were conducted at one location in Georgia (1994) and at two locations in Alabama (1994 and 1995) to evaluate the effects of MSMA or DSMA plus pyrithiobac applied postemergence (POST) in cotton. Pyrithiobac at 0.07 kg ai/ha was applied POST alone or in combination with MSMA at 1.1 kg ai/ha or DSMA at 1.7 kg ai/ha at the pinhead square stage of cotton growth. Cotton was tolerant to the POST applications of pyrithiobac. Adding MSMA or DSMA to pyrithiobac injured cotton similar to MSMA or DSMA applied alone. Plant mapping data indicated that all treatments had no effect on height : node ratio, reproductive or vegetative node production, or square retention at the first or second fruiting position. Cotton maturity response to MSMA and DSMA ranged from no effect to delayed maturity. Adding DSMA to pyrithiobac increased Florida beggarweed and common cocklebur control over pyrithiobac applied alone in 1995 but did not increase control in 1994. Adding DSMA to pyrithiobac increased sicklepod control over pyrithiobac applied alone in the three site years it was rated in the Alabama tests. Where sicklepod is present, the addition of an arsenical herbicide to pyrithiobac will generally increase control but has the potential to delay maturity and decrease cotton yield equal to the arsenical herbicide applied alone.

Field and greenhouse experiments evaluated interactions of soil-applied insecticides and imazaquin and imazethapyr on growth and development of cotton. Imazaquin and imazethapyr were applied PPI at 0 to 6 and 0 to 16 g ae ha−1, respectively, in the greenhouse (plus a no-insecticide control), and 0 to 72 g ha−1 in the field in combination with aldicarb, disulfoton, and phorate (without a no-insecticide control) applied in the seed furrow. Cotton shoot fresh weight in the greenhouse experiment decreased linearly as herbicide rates increased. Greater reductions in shoot fresh weight were noted with imazaquin than with imazethapyr. Compared with no insecticide, the methylcarbamate insecticide aldicarb and the organophosphate insecticides disulfoton and phorate did not affect cotton response to either herbicide. In the field, cotton injury increased while stand, yield, and maturity decreased as herbicide rates increased. Delayed maturity was due to a lower percentage of bolls produced on sympodia from main stem nodes four to nine. Imazaquin caused greater injury, greater reductions in stand, greater delays in maturity, and lower yields than did imazethapyr. Earlier maturity was noted with aldicarb-treated cotton. Compared with aldicarb, disulfoton and phorate did not alter cotton response to imazaquin or imazethapyr.